Hof1 plays a checkpoint-related role in MMS-induced DNA damage response in Candida albicans
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Nucleotide Excision Repair Protein Rad23 Regulates Cell Virulence Independent of Rad4 in Candida albicans
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Critical Role of DNA Checkpoints in Mediating Genotoxic-Stress–induced Filamentous Growth in Candida albicans
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Functional connections between cell cycle and proteostasis in the regulation of Candida albicans morphogenesis - ScienceDirect
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Molecular Microbiology, Microbiology Journal
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Rad53- and Chk1-Dependent DNA Damage Response Pathways Cooperatively Promote Fungal Pathogenesis and Modulate Antifungal Drug Susceptibility
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The Saccharomyces cerevisiae RAD9, RAD17 and RAD24 genes are required for suppression of mutagenic post-replicative repair during chronic DNA damage. - Abstract - Europe PMC
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A J Domain Protein Functions As A Histone Chaperone To, 56% OFF
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Hof1 plays a checkpoint-related role in MMS-induced DNA damage response in Candida albicans
![](https://journals.asm.org/cms/10.1128/msphere.00062-20/asset/3b586e8e-25f3-488c-9de2-499d75b83a08/assets/graphic/msphere.00062-20-f0007.jpeg)
Nucleotide Excision Repair Protein Rad23 Regulates Cell Virulence Independent of Rad4 in Candida albicans
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A Noncanonical DNA Damage Checkpoint Response in a Major Fungal Pathogen
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Analysis of hof1 phenotypes in the absence of Bni1 or Bnr1 function.
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A Noncanonical DNA Damage Checkpoint Response in a Major Fungal Pathogen
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Hof1 plays a checkpoint-related role in MMS-induced DNA damage response in Candida albicans