CAMKIIγ is a targetable driver of multiple myeloma through CaMKIIγ
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Frontiers Deregulated calcium signaling in blood cancer
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PDF] CAMKIIγ is a targetable driver of multiple myeloma through
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Stabilization of the c-Myc Protein by CAMKIIγ Promotes T Cell
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Stabilization of the c-Myc Protein by CAMKIIγ Promotes T Cell
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Recent advances in targeting the “undruggable” proteins: from drug
![](https://www.researchgate.net/publication/319322575/figure/fig4/AS:614260967215104@1523462635000/Activated-CaMKIIg-phosphorylates-FOXO3a-by-directly-or-indirectly-phosphorylating-AKT_Q320.jpg)
Berbamine (BBM) inhibits the growth of T-ALL cells partially by
CAMKIIγ is a targetable driver of multiple myeloma through CaMKIIγ
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Frontiers Strategies to target the cancer driver MYC in tumor cells
![](https://www.researchgate.net/publication/322327985/figure/fig1/AS:613942573412374@1523386724348/Kaplan-Meier-survival-curves-for-progression-free-survival-and-overall-survival-according_Q320.jpg)
Baseline clinical characteristics of the 94 patients with multiple
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Drugging the efferocytosis process: concepts and opportunities
![](https://www.researchgate.net/publication/342914510/figure/fig2/AS:920125679087617@1596386465801/WBC158-exhibited-more-potent-anti-proliferation-activity-than-BBM-partially-through_Q320.jpg)
Overexpression of CAMKIIγ was associated with disease progression
![](https://www.spandidos-publications.com/article_images/or/41/4/OR-41-04-2194-g02.jpg)
In silico analysis identified miRNA‑based therapeutic agents
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Pharmaceutics, Free Full-Text
CAMKIIγ is a targetable driver of multiple myeloma through CaMKIIγ