CAMKIIγ is a targetable driver of multiple myeloma through CaMKIIγ

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Frontiers Deregulated calcium signaling in blood cancer

PDF] CAMKIIγ is a targetable driver of multiple myeloma through

Stabilization of the c-Myc Protein by CAMKIIγ Promotes T Cell

Stabilization of the c-Myc Protein by CAMKIIγ Promotes T Cell

Recent advances in targeting the “undruggable” proteins: from drug

Berbamine (BBM) inhibits the growth of T-ALL cells partially by

CAMKIIγ is a targetable driver of multiple myeloma through CaMKIIγ

Frontiers Strategies to target the cancer driver MYC in tumor cells

Baseline clinical characteristics of the 94 patients with multiple

Drugging the efferocytosis process: concepts and opportunities

Overexpression of CAMKIIγ was associated with disease progression

In silico analysis identified miRNA‑based therapeutic agents

Pharmaceutics, Free Full-Text

CAMKIIγ is a targetable driver of multiple myeloma through CaMKIIγ